Luu triplets biography of barack
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Short Biography
Dr. Schmitt obtained her B.S. in chemistry and biochemistry from Ball State University, where she was a Lewis Stokes Alliances for Minority Participation (LSAMP) Scholar. She obtained her Ph.D. in biochemistry at University of Maryland Baltimore County with Professor Songon An. Her doctoral work was on identifying mechanisms for formation of multienzyme complexes formed by metabolic enzymes. She was a University of California President’s Postdoctoral Fellow at University of California San Diego with Professor Jin Zhang. There, she used genetically encoded reporters for kinase activity to study subcellular regulation of cellular signal transduction. Dr. Schmitt joined the UCLA faculty in 2022. The focus of her laboratory is developing new genetically encoded tools which can be used to study metabolic events in single cells.
Research Interests
The Schmitt Lab is focused on answering the question: How does the cell organize essential processes like metabolism and sig
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DM1
Endocrine Function Over Time in Patients with Myotonic Dystrophy Type 1
Published on Thu, 10/16/2014
Dahlqvist et al
European Journal of Neurology
Dr. John Vissing and his colleagues at the University of Copenhagen recently tracked a group of 68 adults with myotonic dystrophy type 1 (DM1), measuring their endocrine function change over 8 years. The authors examined bloodwork for many endocrine dysfunctions including diabetes (HbA1c blood test), hyperparathyroidism (PTH blood test), and androgen insufficiency (testosterone blood test in men), and found that these dysfunctions became more common over time in people with DM1. The authors recommend that doctors treating people with DM1 should screen for endocrine functions regularly, as the dysfunctions occurs more frequently in DM1 than the general population.
Click here to read the abstract for this study.
Click here for a PDF of this paper.
10/16/2014
Parental Age Effects in
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Abstract
Cardiovascular defects, injuries, and degenerative diseases often require surgical intervention and the use of implantable replacement material and conduits. Traditional vascular grafts made of synthetic polymers, animal and cadaveric tissues, or autologous vasculature have been utilized for almost a century with well-characterized outcomes, leaving areas of unmet need for the patients in terms of durability and long-term patency, susceptibility to infection, immunogenicity associated with the risk of rejection, and inflammation and mechanical failure. Research to address these limitations is exploring avenues as diverse as gene therapy, cell therapy, cell reprogramming, and bioengineering of human tissue and replacement organs. Tissue-engineered vascular conduits, either with viable autologous cells or decellularized, are the forefront of technology in cardiovascular reconstruction and offer many benefits over traditional graft materials, particularly in the potential for